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A close analog of methylphenidate, methylphenidate is a psychostimulant and is also known as Baxtercaine in the United Kingdom. Dopamine reuptake inhibitors and norepinephrine reuptake inhibitors are both inhibited by ethylphenidate. As a result of binding to, and partially blocking, the transporter proteins that generally remove monoamines from synaptic clefts, it effectively increases the levels of norepinephrine and dopamine in the brain.
In addition to acting as a dopamine reuptake inhibitor and norepinephrine reuptake inhibitor, Ethylphenidate CRYSTAL are potent psychostimulants. Through binding to, and partially blocking, the transporter proteins that normally remove those monoamines from the synaptic cleft, this drug effectively boosts norepinephrine and dopamine levels in the brain.
ETHYLPHENIDATE CRYSTALS are similar in structure to methylphenidate (Ritalin) and are available only for research purposes.
ETHYLPHENIDATE CRYSTALS are sold to researchers in powder, crystal, and pellet form for in vitro use only. It is not meant for human consumption.
David Nichols has proposed O-acetyl psilocin as an alternative to psilocybin for pharmacological studies as it is believed to be a prodrug of psilocin.
Synthetic tryptamine 4-ACO-DMT is also known as O-Acetylpsilocin, 4-Acetoxy-DMT, or Psilacetin. This is the acetylated form of the psilocybin mushroom alkaloid psilocin, which is lower in homology than 4-AcO-DET, 4-AcO-MiPT, and 4-AcO-DiPT. Since it is a prodrug of psilocin, David Nichols suggested that it could replace psilocybin in pharmacological studies. Due to its structural similarity to psilocin and psilocybin, it has the same subjective effect profile. So, 4-AcO-DMT can replace psilocybin mushrooms in this way.
On January 16, 1963, Sandoz Ltd. patented 44-ACO-DMT and several other esters of psilocin through Albert Hofmann & Franz Troxler. 4-ACO-DMT had a limited history of use before its release as a gray area compound on the online research chemical market.
4-ACO-DMT ‘s psychedelic effects are believed to come from its efficacy at the 5-HT2A receptor as a partial agonist. However, the role of these interactions and how they result in the psychedelic experience continues to remain elusive.
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